Don’t take our word for it. Here’s every fact on this site, with its evidence grade, its caveats, and a link to the source, so you can check us.
We start you on the strong ones: findings that have been replicated or come from one large, well-controlled study. Moderate and weak facts are here too, labeled for what they are. Use “Link to this fact” to share a single card.
Children and pregnancy / Measured in people or real products
In the same 2018 NGO test, a children's shampoo had 24 unlabeled chemicals flagged for health hazards (17 on the fragrance-industry list), and a baby shampoo had 6.
Weak evidence: Counts, not amounts; single samples; not peer reviewed; not a risk ranking.
Source: BCPP 2018 Link to this fact
Children and pregnancy / Government agency
In animal studies, a pregnant female's exposure to an endocrine-disrupting chemical can affect not only her offspring but her offspring's offspring, because developmental exposure can alter reproductive-cell programming that carries across generations.
Weak evidence: Evidence for these transgenerational effects comes from animal studies cited in the report; no multigenerational human dataset is cited for this specific claim.
Source: WHO/UNEP 2012 Link to this fact
Children and pregnancy / Other evidence
A 2020 review states that endocrine-disrupting chemicals found in the placenta (pesticides, personal care product chemicals, flame retardants, BPA, PCBs) could be associated with fetal growth restriction, thyroid dysfunction, and neurological disorders, but the mechanisms are largely unknown.
Weak evidence: Association stated through a single cited review; mechanisms rest largely on animal and small human studies; not a demonstrated causal risk.
Source: Street 2020, International Journal of Molecular Sciences Link to this fact
Children and pregnancy / Other evidence
In the same Korean first-trimester cohort, triclosan, benzophenone-3 and methyl-, ethyl-, propyl- and butylparaben (personal-care-product chemicals) were not associated with TSH or with the oxidative-stress markers MDA and 8-OHdG.
Weak evidence: A null from one small cohort with one spot urine per woman; it does not show safety, and it contradicts earlier cohorts the authors cite (propylparaben, BPS).
Source: Kim 2025, Environmental Analysis Health and Toxicology Link to this fact
Children and pregnancy / Other evidence
The authors write that oxidative stress mediates EDC effects on thyroid function, but their data show no such link: none of the three TSH-associated chemicals was associated with MDA or 8-OHdG, and no mediation analysis was run.
Weak evidence: Absence of association in one underpowered cohort is not evidence against a ROS pathway either; the point is that the paper did not test it.
Source: Kim 2025, Environmental Analysis Health and Toxicology Link to this fact
Children and pregnancy / Other evidence
In the ECHO cohorts, prenatal paraben and benzophenone levels showed stronger associations with eczema in girls than boys (girls: parabens OR 1.21, 1.04-1.42; benzophenones 1.18, 1.00-1.38; bisphenol S 1.21, 1.03-1.43), a pattern the authors call consistent across chemicals.
Weak evidence: Stratified estimates with no correction for multiple tests and no interaction p reported for the eczema comparisons in the main text; subgroup finding.
Source: Miller 2025 (ECHO), Environment International Link to this fact
Children and pregnancy / Other evidence
The positive estimates for the pregnancy chemical mixture came mainly from the Cincinnati HOME cohort, which had higher exposure levels (behavior symptom index about 5 points higher per quartile, CI -1.6 to 11.6), while the Canadian MIREC cohort showed essentially none (-0.06, CI -1.65 to 1.52).
Weak evidence: Small HOME subsample (n=139), wide CIs including zero, results as reported in the text rather than checked in the supplement.
Source: Puvvula 2026, Environ Int, Environment International Link to this fact
Children and pregnancy / Review of other studies
The review treats thyroid disruption by endocrine-disrupting chemicals in early pregnancy as a route to fetal neurodevelopmental harm, but it presents no test of that chain and does not cite the Norwegian MoBa analysis in which maternal thyroid hormones did not mediate the DEHP and child ADHD association (indirect OR 1.00).
Weak evidence.
Caveats
Engel 2018 used one mid-pregnancy sample, total hormones, no thyroid antibodies and one outcome, so it does not rule out a thyroid route; the review's chain is plausible but untested.
Children and pregnancy / Other evidence
A 2026 systematic review of 127 studies called the link between early-life exposure to endocrine-disrupting chemicals (phthalates, BPA, pesticides, PBDEs) and ADHD 'consistent', but it did no meta-analysis, gave no effect sizes or per-chemical grades, and says causality is not established.
Weak evidence: Ungraded wording; the EPA systematic review grades phthalate-ADHD/behavior evidence slight or indeterminate for most pairs; Table 1 details do not all match the primaries.
Source: Wu 2026, Biological Psychiatry Global Open Science Link to this fact
Information, not medical advice. See also: myths we won’t tell you.